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- Neuroplasticity
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epression is a common, chronic, and often dis-
abling psychiatric illness, which is estimated to affect 5%
to 10% of the population. It frequently appears in early
life, has a chronic course, and is considered a risk factor
for other medical illnesses, such as coronary vascular dis-
ease, diabetes, and osteoporosis.This is not altogether sur-
prising given the extensive bidirectional mind-body
interactions mediated via the autonomic nervous system,
immune system, and a host of neuroendocrine factors.
According to the World Health Organization (WHO),
depression is the leading global cause of years of life lived
with disability and the fourth leading cause of disability-
adjusted life-years. Disability-adjusted life-years is
defined as the reduction in an individuals productive life,
and takes into account premature mortality.
1,2
Considering the high morbidity and mortality associated
with depression, it is unfortunate that the psychological
and neurobiological underpinnings of depression have
not been specifically defined.Although major depression
is currently diagnosed by means of a diagnostic system
(Diagnostic and Statistical Manual of Mental Health
Disorders, Fourth Edition [DSM-IV])
based upon phe-
nomenology, this disorder most likely embodies a het-
erogeneous set of disorders with multiple causes.
Therefore, one of the major goals of current and future
research on depression is the development of a diagnos-
tic system based on etiology.
3
This goal is becoming increasingly closer to reality due to
recent progress in the identification of neural circuits,
neurochemicals, and signal transduction mechanisms
2 1 7
C l i n i c a l r e s e a r c h
D
Copyright © 2004 LLS SAS. All rights reserved
www.dialogues-cns.org
Cellular plasticity and resilience and the
pathophysiology of severe mood disorders
Dennis S. Charney, MD; Georgette DeJesus, MD; Husseini K. Manji, MD
Keywords:
mood disorder; depression; neuroplasticity; stress; resilience; brain
morphology
Author affiliations:
National Institute of Mental Health, Bethesda, Md, USA
Address for correspondence:
Prof Dennis S. Charney, National Institute of
Mental Health, 15K North Drive, Room 101, Bethesda, MD 20892-2670, USA
(e-mail: charneyd@nih.gov)
Recent advances in the identification of the neural cir-
cuits, neurochemicals, and signal transduction mecha-
nisms involved in the pathophysiology and treatment of
mood disorders have led to much progress toward
understanding the roles of genetic factors and psy-
chosocial stressors. The monoaminergic neurotransmit-
ter systems have received the most attention, partly
because of the observation that effective antidepressant
drugs exert their primary biochemical effects by regu-
lating intrasynaptic concentrations of serotonin and nor-
epinephrine. Furthermore, the monoaminergic systems
are extensively distributed throughout the network of
limbic, striatal, and prefrontal cortical neuronal circuits
thought to support the behavioral and visceral manifes-
tations of mood disorders. Increasing numbers of neu-
roimaging, neuropathological, and biochemical studies
indicate impairments in cellular plasticity and resilience
in patients who suffer from severe, recurrent mood dis-
orders. In this paper, we describe studies identifying pos-
sible structural, functional, and cellular abnormalities
associated with depressive disorders, which are poten-
tially the cellular underpinnings of these diseases. We
suggest that drugs designed to enhance cellular plastic-
ity and resilience, and attenuate the activity of mal-
adaptive stress-responsive systems, may be useful for the
treatment of severe mood disorders.
© 2004, LLS SAS
Dialogues Clin Neurosci
. 2004;6:217-225.